Bart syndrome (BS) was named after Bartter et al. first reported two cases of hypokalemia metabolic alkalosis, hyperaldosteronism, normal blood pressure, and renal histological examination showed hypertrophy of the paraglomerular organs in 1962. By the 1990s, more than 200 cases have been reported, and some patients may have a family history. The pattern is autosomal recessive inheritance. Clinical data shows that although the cases named after BS are characterized by the loss of kidney salt, hypokalemia metabolic alkalosis, increased blood renin and aldosterone levels, and normal blood pressure, they have other clinical manifestations and laboratory tests.