Hyperactivity of neurons in specific areas of the brain is thought to be an early disturbance of Alzheimer's disease. The long-term existence of excitatory neurotransmitters in the vicinity of active neurons leads to pathological over stimulation of these neurons, which has a serious impact on the loss of learning and memory in patients with Alzheimer's disease. Studies have shown that Alzheimer's patients have a large number of β - amyloid proteins called plaques in their brains. Amyloid β - protein (10-20) is a fragment of β - amyloid protein, which is a peptide composed of 11 amino acids. This sequence starts from the 10th amino acid at the N-terminal and ends at the 20th amino acid at the N-terminal. Compared with β - amyloid protein, the production process of Amyloid β - protein (10-20) is relatively simple and has better solubility.
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